NFAT1在脊髓背角小胶质细胞中调节神经病理性疼痛机制的研究（所属项目其他成果）
1.G protein-coupled receptor GPR151 is involved in trigeminal neuropathic pain through the induction of G beta gamma/extracellular signal-regulated kinase-mediated neuroinflammation in the trigeminal ganglion（G蛋白偶联受体GPR151通过诱导G β-γ/细胞外信号调节激酶介导的神经炎症而参与三叉神经痛）
2.TLR8 and its endogenous ligand miR-21 contribute to neuropathic pain in murine DRG （TLR8（Toll样受体）及其内源性配体miR-21对小鼠DRG神经性疼痛的作用）
3.Chemokine receptor CCR2 contributes to neuropathic pain and the associated depression via increasing NR2B-mediated currents in both D1 and D2 dopamine receptor-containing medium spiny neurons in the nucleus accumbens shell（趋化因子受体CCR2通过增加伏隔核壳中含有D1和D2多巴胺受体的中棘神经元中NR2B介导的电流，促进神经性疼痛和相关抑郁症）
4.趋化因子CXCL10作为神经病理性疼痛生物标记物的研究
5.ZNF382 controls mouse neuropathic pain via silencer-based epigenetic inhibition of Cxcl13 in DRG neurons （ZNF382（锌指蛋白转录因子）通过基于沉默者的表观遗传抑制DRG神经元中的Cxcl13控制小鼠神经性疼痛）
6.CXCL13通过CXCR5激活星形胶质细胞中JNK参与痛觉过敏
7.Chemokines in chronic pain: cellular and molecular mechanisms and therapeutic potential（慢性疼痛中的趋化因子：细胞和分子机制以及治疗潜力）
8.MMP24 Contributes to Neuropathic Pain in an FTO-Dependent Manner in the Spinal Cord Neurons （基质金属蛋白酶24 在脊髓神经元中依赖于FTO（肥胖调节基因，一种RAN 6-甲基腺苷（m6A）脱甲基酶，促进肿瘤细胞的糖酵解，抑制T细胞活性，影响功能，帮助肿瘤实现免疫逃避）方式促进神经性疼痛）
9.慢性疼痛的分子机制和镇痛新靶点研究
10.Increased CXCL13 and CXCR5 in Anterior Cingulate Cortex Contributes to Neuropathic Pain-Related Conditioned Place Aversion （前扣带回中CXCL13和CXCR5的增加有助于神经性疼痛相关的条件性位置规避）
11.Demethylation of G-Protein-Coupled Receptor 151 Promoter Facilitates the Binding of Kruppel-Like Factor 5 and Enhances Neuropathic Pain after Nerve Injury in Mice （G蛋白偶联受体151启动子的去甲基化促进Kruppel样因子5的结合并增强小鼠神经损伤后的神经性疼痛）
12.Intravenous Administration of Triptonide Attenuates CFA-Induced Pain Hypersensitivity by Inhibiting DRG AKT Signaling Pathway in Mice （静脉注射雷公藤内酯醇通过抑制DRG AKT信号通路减轻CFA诱导的小鼠疼痛超敏）
13.Spinal CXCL9 and CXCL11 are not involved in neuropathic pain despite an upregulation in the spinal cord following spinal nerve injury （尽管在脊髓神经损伤后，脊髓中的 CXCL9和 CXCL11表达上调，但它们并不参与神经性疼痛）
